File:MCS-hypoxia-model.png
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A model of Multiple Chemical Sensitivity. The putative pathways elicited by sensitizing molecules in the activation of vicious NO/NOO− cycle, stimulating lactate production and tissue hypoxia leading to acidosis and compensatory pulmonary hyperventilation, and eventually form specific exhaled VOCs profiles that we recorded in the present study and are graphically presented in Figure 5.
Author: (or citation)[edit | edit source]
Mazzatenta, Andrea; Pokorski, Mieczyslaw; Giulio, Camillo Di (2021). "Volatile organic compounds (VOCs) in exhaled breath as a marker of hypoxia in multiple chemical sensitivity". Physiological Reports. 9 (18): e15034. doi:10.14814/phy2.1503.
Source: (e.g. internet address)[edit | edit source]
Mazzatenta, A., Pokorski, M., & Di Giulio, C. (2021). Volatile organic compounds (VOCs) in exhaled breath as a marker of hypoxia in multiple chemical sensitivity. Physiological Reports, 9, e15034. https://doi.org/10.14814/phy2.15034[1] Figure 5.
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This file is licensed under the Creative Commons Attribution 4.0 International license. | ||
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- ↑ Mazzatenta, Andrea; Pokorski, Mieczyslaw; Giulio, Camillo Di (2021). "Volatile organic compounds (VOCs) in exhaled breath as a marker of hypoxia in multiple chemical sensitivity". Physiological Reports. 9 (18): e15034. doi:10.14814/phy2.15034. ISSN 2051-817X. PMC 8449310. PMID 34536058.
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current | 17:55, January 7, 2022 | 500 × 276 (123 KB) | Notjusttired (talk | contribs) | ==== Title: (or description) ==== A model of Multiple Chemical Sensitivity. The putative pathways elicited by sensitizing molecules in the activation of vicious NO/NOO− cycle, stimulating lactate production and tissue hypoxia leading to acidosis and... |
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